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SARMs are already discerning by definition, but research confirms that RAD-140 binds particularly well to the androgen receptors in bone and muscle, so that its effect is only modest with regard to testosterone. In contrast to its immediate effect in reducing testosterone, SAMs have an even greater effects when taken orally (e.g., in combination with the other forms of rT3); these effects are enhanced by other nonhormonal factors, e.g., dietary supplements.
While SAMs may be able to reduce testosterone levels significantly, we find little evidence that they exert other effects with regard to other male gender steroid hormones other than testosterone. Indeed, in the study by Reeder et al. (2003) and by Fonvé et al. (1985), neither SAMs nor their salts or rT3 had major effects on the aromatase enzyme, which converts androsterone to estrogens. Furthermore, it is unclear whether SAMs reduce aromatase activity in vivo, because one study found a marked increase in aromatase activity in the testes of rats exposed to SAMs (Barrett et al. 1998), whereas a follow-up study showed a significant decrease (in vivo: −1.9 nmol/g protein; 1 h: 2.5 pmol/g protein) in the testes of the same animals (Barrett et al. 2000). SAMs did not significantly reduce testosterone levels in human males, and they did not significantly suppress estradiol levels in females.
Although SAMs are present in the serum of many healthy adults, it has also been shown that a deficiency in SAMs can lead to significant problems in healthy men and women with regard to the functioning of their male sex and/or their estrogen levels. A severe vitamin B12 deficiency (and its effects thereon) are common to women with low testosterone and high estrogen (Dwyer 1992). In contrast, men with low testosterone and low estradiol levels tend to be vitamin B12 deficient but not vitamin B12 deficient (Barrett 1996). In normal males, SAMs are essential for vitamin B12 absorption, and for this reason SAM supplementation is generally regarded as a necessary component of a healthy diet (Barrett 1992).
Thus, because SAMs are not widely distributed in humans, it is possible that they may play an important and often unrecognized role in the development of the physical and psychological characteristics of men with low testosterone and low estradiol levels. Moreover, such men may have greater difficulty in maintaining their normal levels of hormones than normal men whose testosterone levels and estradiol levels are well above those of normal males (
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